By Philip M. Parker
In March 2001, the nationwide Institutes of healthiness issued the subsequent caution: "The variety of sites providing health-related assets grows each day. Many websites supply useful info, whereas others could have details that's unreliable or misleading." additionally, as a result of swift elevate in Internet-based info, many hours will be wasted looking, settling on, and printing. on the grounds that in basic terms the smallest fraction of data facing Alzheimer sickness is listed in se's, reminiscent of www.google.com or others, a non-systematic method of web learn may be not just time eating, but additionally incomplete. This booklet used to be created for doctors, scholars, and individuals of most of the people who are looking to behavior scientific study utilizing the main complicated instruments to be had and spending the smallest amount of time doing so.
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Extra resources for Alzheimer Disease - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers
If we can demonstrate significant effects of CoQ10 and L-NIL, this could lead to rapid development of new therapies for slowing the progression of AD. ; Assistant Professor; Massachusetts General Hospital 55 Fruit St Boston, Ma 02114 Timing: Fiscal Year 2006; Project Start 15-FEB-2006; Project End 31-DEC-2010 Summary: (provided by applicant): Alzheimer's disease (AD) is a devastating neurological illness with no known cure, yet a central hypothesis implicating oxidative stress as a cause of the disease has been postulated for more than a decade.
We have carefully selected women at high risk for dementia and by careful screening we have selected to reduce the risk of the known estrogen related adverse outcomes. We propose to continue this 5 year multicenter, randomized, double blind placebo controlled trial of estrogens (Premarin or Prempro) to assess its efficacy to prevent memory loss and dementia in 900 healthy, elderly women greater than or equal to age 65 with a family history of AD recruited at 27 sites. Subjects will be assessed at 6 month intervals for safety and compliance and at annual intervals for cognitive outcomes.
We will test our hypothesis with three specific aims. Aim 1: Assess for group differences in CR fitness in a cohort of nondemented controls and early-stage AD participants. Aim 2: Evaluate the relationship of CR fitness and hippocampal volume in non-demented aging and early-stage AD. Aim 3: Assess CR fitness in relation to MRI markers of brain aging: atrophy, white matter lesions, and infarcts. This preliminary, multidisciplinary study will generate crosssectional pilot data assessing the relation between CR fitness, brain aging and ADrelated brain injury.