New PDF release: Advanced examples in physics

By Allen. Arthur Ormiston

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BMC Neurosci. 2009;10:7. 16. Dietrich HH, Dacey RG Jr. Molecular keys to the problems of cerebral vasospasm. Neurosurgery 2000;46(3):517 30. 17. Dorsch NW. Cerebral arterial spasm a clinical review. Br J Neu rosurg. 1995;9:403 12. 18. Dumont AS, Dumont RJ, Chow MM, Lin CL, Calisaneller T, Ley KF, et al. Cerebral vasospasm after subarachnoid hemorrhage: putative role of inflammation. Neurosurgery 2003;53(1):123 33. 19. Endo H, Nito C, Kamada H, Yu F, Chan PH. Reduction in oxida tive stress by superoxide dismutase overexpression attenuates acute brain injury after subarachnoid hemorrhage via activation of Akt/glycogen synthase kinase 3beta survival signaling.

Signal transduction mechanisms play pivotal roles in such contraction. Each role of kinases or phosphorylated proteins has been clarified, but it is not clear how they integrate or regulate each other in the development and maintenance of cerebral vasospasm. Interrelation of the roles of kinases or phosphorylated proteins should be overviewed for understanding the mechanisms of long-lasting arterial constriction [3]. The purpose of this article is to introduce and review just some parts of such interrelations.

Tseng MY, Hutchinson PJ, Richards HK, Czosnyka M, Pickard JD, Erber WN, et al. Acute systemic erythropoietin therapy to reduce delayed ischemic deficits following aneurysmal subarachnoid hemorrhage: a Phase II randomized, double blind, placebo con trolled trial. Clinical article. J Neurosurg. 2009;111:171 80. Agrawal A, Patir R, Kato Y, Chopra S, Sano H, Kanno T. Role of intraventricular sodium nitroprusside in vasospasm secondary to aneurysmal subarachnoid haemorrhage: a 5 year prospective study with review of the literature.

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